BioAmpMax
Together they are designed to:
- Amplify glucose disposal & insulin sensitivity through direct AMPK activation (ATX-304) and NNMT inhibition (5-Amino-1MQ), plus enhanced insulin signaling via myo- and D-chiro-inositol.
- Improve lipid & endothelial health by reducing adiposity, supporting healthy LDL-C, lowering blood pressure, and boosting nitric-oxide–mediated vascular function.
- Enhance mitochondrial energy metabolism via AMPK-driven biogenesis (ATX-304) and NAD⁺ preservation (5-Amino-1MQ), fostering efficient fuel utilization.
- Support inositol-mediated insulin signaling & glycogen synthesis with myo-inositol and D-chiro-inositol in a physiological ratio for optimized glucose handling.
- Restore redox balance & quell inflammation through glutathione replenishment and antioxidant activity (N-acetylcysteine).
These complementary mechanisms make BioAmpMax a promising research candidate for studies of metabolic syndrome, cardiometabolic wellness, and healthy aging.
BioAmpMax Structure
| Ingredient | Dose | Key Actions |
| ATX-304 (OS-01) | 100 mg | Pan-AMPK activation; mimics exercise to improve insulin sensitivity & glucose uptake [1] |
| 5-Amino-1MQ | 75 mg | NNMT inhibition; raises NAD⁺, enhances fat oxidation & insulin responsiveness [2] |
| Myo-Inositol | 1000 mg | Insulin-signaling mediator; lowers fasting insulin & HOMA-IR in humans [3][6] |
| D-Chiro-Inositol | 20 mg | Insulin-mimetic isomer; promotes glycogen synthesis & post-prandial glucose disposal [5] |
| Chromium Picolinate | 200 µg | Insulin-receptor cofactor; modestly improves glycemic control & lipid parameters [7][8] |
| N-Acetylcysteine (NAC) | 600 mg | Glutathione precursor; reduces oxidative stress & inflammation, enhances endothelial NO [9] |
Research Areas
- Glucose Disposal & Insulin Sensitivity
- Lipid & Endothelial Support
- Mitochondrial & Energy Metabolism
- Inositol Signaling & Glycogen Synthesis
- Redox Balance & Anti-inflammatory Support
